SIX1 (D4A8K) Rabbit mAb

SIX1 (D4A8K) Rabbit mAb Suppliers list
Company Name: Cell Signaling Technology Inc  
Tel: 21-80243558 86218024
Email: info@cellsignal.cn
Company Name: Guangzhou Bingheng Biotechnology Co., Ltd.  
Tel: 13631342304
Email:
Company Name: Hangzhou Novas Biotechnology Co., Ltd.  
Tel: 15264006066
Email:
Company Name: Shanghai Tuoran Biotechnology Co., Ltd.  
Tel: 18621916228
Email:
Company Name: Shanghai Gaochuang Chemical Technology Co., Ltd.  
Tel: 021-37695331 13918520284
Email: gaochem@163.com
SIX1 (D4A8K) Rabbit mAb Basic information
Source Reactivity Background References
Product Name:SIX1 (D4A8K) Rabbit mAb
Synonyms:SIX1 (D4A8K) Rabbit mAb
CAS:
MF:
MW:0
EINECS:
Product Categories:
Mol File:Mol File
SIX1 (D4A8K) Rabbit mAb Structure
SIX1 (D4A8K) Rabbit mAb Chemical Properties
Safety Information
MSDS Information
SIX1 (D4A8K) Rabbit mAb Usage And Synthesis
SourceRabbit
ReactivityHuman;Mouse;Rat;Monkey
BackgroundSine oculis homeobox proteins belong to a family of evolutionarily conserved transcription factors discovered in Drosophila mutant screens for embryonic eye development genes. The prototypical family member was named for eyeless embryos carrying mutations in a gene highly conserved among vertebrates, including humans. A total of six family members have been identified in vertebrates. Each SIX protein contains a homeobox nucleic acid recognition domain with a DNA-binding helix-turn-helix motif and an adjacent SIX domain, which may be involved in regulating protein-protein interactions. In addition to their critical functions during embryonic organogenesis, research studies suggest that SIX proteins play additional roles in postnatal cell cycle regulation, with potentially important implications in tumorigenesis.In contrast to the Drosophila ortholog, the vertebrate SIX1 gene product does not play a critical role in embryonic eye development. Vertebrate SIX1 is required for development of mesoderm- and neural crest-derived lineages, and male reproductive tissues. SIX1 has also been shown to regulate transcription of MyoD in adult muscle progenitor cells during postnatal muscle development. A mechanistic role for SIX1 in cell cycle regulation is supported by research studies showing increased SIX1 expression in various cancer subtypes, including breast, ovarian, and hepatocellular carcinoma.
References[1] Kumar, J.P. (2009) Cell Mol Life Sci 66, 565-83.
[2] Fischbach, K.F. and Technau, G. (1984) Dev Biol 104, 219-39.
[3] Fischbach, K.F. and Heisenberg, M. (1981) Proc Natl Acad Sci U S A 78, 1105-9.
[4] Boucher, C.A. et al. (1996) Genomics 33, 140-2.
[5] Pignoni, F. et al. (1997) Cell 91, 881-91.
[6] Ford, H.L. et al. (1998) Proc Natl Acad Sci U S A 95, 12608-13.
[7] Coletta, R.D. et al. (2004) Proc Natl Acad Sci U S A 101, 6478-83.
[8] Oliver, G. et al. (1995) Development 121, 4045-55.
[9] Kaiser, R. et al. (2007) Am J Med Genet A 143A, 2185-8.
[10] Fujimoto, Y. et al. (2013) Dev Cell 26, 416-30.
SIX1 (D4A8K) Rabbit mAb Preparation Products And Raw materials
Tag:SIX1 (D4A8K) Rabbit mAb Related Product Information