MIPS-521

MIPS-521 Suppliers list
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MIPS-521 Basic information
Product Name:MIPS-521
Synonyms:MIPS-521;5-bis(trifluoromethyl)phenyl]thiophen-3-yl}(4-chlorophenyl)methanone;{2-Amino-4-[3,5-bis(trifluoromethyl)phenyl]thiophen-3-yl}(4-chlorophenyl)methanone;A1AR,inhibit,MIPS-521,pain,Adenosine Receptor,MIPS 521,MIPS521,Inhibitor,P1 receptor;Methanone, [2-amino-4-[3,5-bis(trifluoromethyl)phenyl]-3-thienyl](4-chlorophenyl)-;MIPS521, 10 mM in DMSO
CAS:1146188-19-3
MF:C19H10ClF6NOS
MW:449.8
EINECS:
Product Categories:
Mol File:1146188-19-3.mol
MIPS-521 Structure
MIPS-521 Chemical Properties
Melting point 178-180 °C(Solv: ligroine (8032-32-4))
Boiling point 516.3±50.0 °C(Predicted)
density 1.481±0.06 g/cm3(Predicted)
storage temp. Store at -20°C
solubility DMSO : 25 mg/mL (55.58 mM; ultrasonic and warming and heat to 60°C)
pka-2.49±0.10(Predicted)
form Solid
color Light yellow to green yellow
Safety Information
MSDS Information
MIPS-521 Usage And Synthesis
UsesMIPS521 is a positive allosteric modulator of adenosine A1 receptor (A1AR). MIPS521 also has a lower A1R allosteric affinity (pKB=4.95; KB=11μM). MIPS521 exhibits pain-relieving effects in vivo through modulation of the increased levels of endogenous adenosine[1][2].
Biological ActivityMIPS521 is a positive allosteric modulator of adenosine A1 receptor (A1AR). MIPS521 also has a lower A1R allosteric affinity (pKB=4.95). MIPS521 exhibits pain-relieving effects in vivo[1][2]. MIPS521 (compound 13o) (3-10 μM) improves the ability of R-PIA to promote A1AR-mediated ERK1/2 phosphorylation[1].MIPS521 (0.3-30 μM; pretreament for 10 min, co-treatment for 30 min) produces a concentration-dependent potentiation of signalling by ADO in an inhibition of cAMP assay (expressed as a percentage of the inhibition of 3 μM forskolin-mediated cAMP) in CHO cells[2]. MIPS521 (1-30 μg in 10 μL; intrathecal administration) reverses mechanical hyperalgesia in rats, promoting robust antinociception[2].MIPS521 (10 μg in 10 μL; intrathecal administration) significantly reduces spontaneous pain in a conditioned place preference model[2].MIPS521 (1-30 μg in 10 μL; intrathecal administration) reduces eEPSCs in spinal cord from nerve-injured rats, with a pEC50 of 6.9. The maximum MIPS521-induced decrease in synaptic current amplitude is significantly greater in nerve-injured rats than in sham surgery controls[2].
in vivo

MIPS521 (1-30 μg in 10 μL; intrathecal administration) reverses mechanical hyperalgesia in rats, promoting robust antinociception[2].
MIPS521 (10 μg in 10 μL; intrathecal administration) significantly reduces spontaneous pain in a conditioned place preference model[2].
MIPS521 (1-30 μg in 10 μL; intrathecal administration) reduces eEPSCs in spinal cord from nerve-injured rats, with a pEC50 of 6.9. The maximum MIPS521-induced decrease in synaptic current amplitude is significantly greater in nerve-injured rats than in sham surgery controls[2].

Animal Model:Male and female Sprague-Dawley rats (7-12 weeks) were performed a partial nerve ligation (PNL) or sham surgery[2]
Dosage:1, 3, 10, 30 μg in 10 μL
Administration:Intrathecal administration
Result:Reduced eEPSCs in spinal cord from nerve-injured rats and reversed mechanical hyperalgesia.
IC 50A1AR
References[1]. Aurelio L, et, al. Allosteric modulators of the adenosine A1 receptor: synthesis and pharmacological evaluation of 4-substituted 2-amino-3-benzoylthiophenes. J Med Chem. 2009 Jul 23;52(14):4543-7. [2]. Draper-Joyce CJ, et, al. Positive allosteric mechanisms of adenosine A 1 receptor-mediated analgesia. Nature. 2021 Sep;597(7877):571-576.
MIPS-521 Preparation Products And Raw materials
Tag:MIPS-521(1146188-19-3) Related Product Information
CTEP Derivative 2-[(Cyclopropylsulfonyl)amino]-N-(2-ethoxyphenyl)benzamide cholesta-3,5-dien-7-one 1-(4-Chlorophenyl)-3-[3-[6-(pyrrolidin-1-yl)pyridin-2-yl]phenyl]urea Methanone, [2-amino-4-[3-(trifluoromethyl)phenyl]-3-thienyl]phenyl- CGS 21680A